Publication & Citation Trends
Publications
0 total
Disease-associated calmodulin variants impair regulation of neuronal voltage-gated calcium channels.
Cited by 0
Semantic Scholar
Elucidating the role Cav1.2 dysfunction in the pathogenesis of timothy syndrome using iPSC-derived neurons. OA
Cited by 2
Semantic Scholar
CACNA1C-Related Channelopathies.
Cited by 18
Semantic Scholar
Calmodulin Mutations in Human Disease OA
Cited by 39
Semantic Scholar
Induced pluripotent stem cell-derived cardiomyocytes enable the development of new therapeutic strategies for the treatment of LQT8. OA
Cited by 0
Semantic Scholar
Fibroblast growth factor homologous factors serve as a molecular rheostat in tuning arrhythmogenic cardiac late sodium current OA
Cited by 20
Semantic Scholar
Antisense oligonucleotides as a treatment strategy for timothy syndrome OA
Cited by 1
Semantic Scholar
LQT8 mutations induce action potential prolongation and arrhythmia in induced pluripotent stem cell derived cardiomyocytes OA
Cited by 0
Semantic Scholar
Research Topics
Ion channel regulation and function
(69)
Cardiac electrophysiology and arrhythmias
(61)
Neuroscience and Neural Engineering
(16)
Cardiomyopathy and Myosin Studies
(11)
Neuroscience and Neuropharmacology Research
(10)
Affiliations
University of North Carolina at Chapel Hill
University of Maryland, Baltimore
Merck & Co., Inc., Rahway, NJ, USA (United States)
Mayo Clinic
Johns Hopkins University