Publication & Citation Trends
Most Cited Works
Publications
13 total
216 (PB204): The mechanistic basis of both deep and durable antitumor activity by combinatorial inhibition of MAT2A and PRMT5 in MTAP-deleted tumors
Cited by 1
OpenAlex
Abstract 1644: Dual inhibition of MAT2A and PRMT5 delivers synergistic anti-tumor responses in preclinical models of <i>MTAP</i>-deleted cancer
Cited by 5
OpenAlex
Abstract 1637: MAT2A inhibition in <i>MTAP-/-</i> tumors confers mechanistic vulnerabilities to multiple clinically actionable synthetic lethal drug combinations
Cited by 2
OpenAlex
Abstract 1637: MAT2A inhibition in MTAP-/- tumors confers mechanistic vulnerabilities to multiple clinically actionable synthetic lethal drug combinations
Cited by 4
Semantic Scholar
Abstract 1278: MAT2A inhibitor, IDE397, displays broad anti-tumor activity across a panel of <i>MTAP</i>-deleted patient-derived xenografts
Cited by 6
OpenAlex
Kinetics of Human Mutant Tau Prion Formation in the Brains of 2 Transgenic Mouse Lines PDF
Cited by 44
OpenAlex
Novel compounds lowering the cellular isoform of the human prion protein in cultured human cells
Cited by 25
OpenAlex
Research Topics
Prion Diseases and Protein Misfolding
(9)
Cancer-related gene regulation
(4)
Epigenetics and DNA Methylation
(3)
Neurological diseases and metabolism
(2)
Parkinson's Disease Mechanisms and Treatments
(1)
Frequent Co-Authors
Affiliations
University of California, San Francisco
German Center for Neurodegenerative Diseases
Ideaya Biosciences (United States)
Institute for Neurodegenerative Disorders