Publication & Citation Trends
Publications
0 total
Protein phosphatase 2A contributes to the cardiac dysfunction induced by endotoxemia OA
Cited by 27
Semantic Scholar
ctivation of p 38 Mitogen-Activated rotein Kinase Contributes to the Early ardiodepressant Action of Tumor Necrosis Factor
Cited by 0
Semantic Scholar
Activation of p38 mitogen-activated protein kinase contributes to the early cardiodepressant action of tumor necrosis factor. OA
Cited by 59
Semantic Scholar
Involvement of the nicotinamide adenosine dinucleotide phosphate oxidase isoform Nox2 in cardiac contractile dysfunction occurring in response to pressure overload. OA
Cited by 151
Semantic Scholar
Abstract 1444: Reversal of Sepsis-induced Contractile Dysfunction by Targeting Cardiac TnI Phosphorylation
Cited by 0
Semantic Scholar
P38-MAPK mediates the early negative inotropic effect of tumour necrosis factor-alpha (TNF). Evidence of synergy between a direct negative inotropic effect and coronary constriction
Cited by 0
Semantic Scholar
Regulation of cardiac contractile function by troponin I phosphorylation. OA
Cited by 428
Semantic Scholar
Protection against endotoxemia‐induced contractile dysfunction in mice with cardiac‐specific expression of slow skeletal troponin I
Cited by 93
Semantic Scholar
Research Topics
Cardiac electrophysiology and arrhythmias
(9)
Cardiomyopathy and Myosin Studies
(9)
Ion channel regulation and function
(6)
Cardiac Fibrosis and Remodeling
(6)
Cardiac Ischemia and Reperfusion
(4)
Affiliations
University of Leeds
Royal Adelaide Hospital
St Thomas' Hospital
Harvard University
King's College London