Publication & Citation Trends
Publications
0 total
The Roles of A Proliferation-Inducing Ligand (APRIL) and B-Cell Activating Factor (BAFF) in IgA Nephropathy.
Cited by 0
Semantic Scholar
Genome-Wide Association Study (GWAS) of Serum Galactose-Deficient IgA1 Uncovers Shared Genetic Determinants with IgAN
Cited by 0
Semantic Scholar
Dysregulated Expression of Genes Encoding Virulence Factors among Metronidazole-Resistant and Sensitive Trichomonas Vaginalis Isolates
Cited by 0
Semantic Scholar
Emerging Biochemical and Immunologic Mechanisms in the Pathogenesis of IgA Nephropathy.
Cited by 1
Semantic Scholar
NF-kB Inhibitor Enhances Galactose Deficiency of IgA1 and Modulates Activation of Multiple Transcription Factors in Immortalized B Cells from Patients with IgAN and Healthy Control Participants
Cited by 0
Semantic Scholar
Fluid Flow Shear Stress-Induced Podocyte Glycosylation Changes: Implications for Glomerular Function
Cited by 0
Semantic Scholar
Cell-Surface Glycosylation Identifies Subpopulations of B Cells in IgA Nephropathy with Distinct Signaling Corresponding to the Production of the Main Autoantigen, Galactose-Deficient IgA1
Cited by 0
Semantic Scholar
#1261 A small-molecule inhibitor of Bruton's tyrosine kinase reduces production of galactose-deficient IgA1 in IgA nephropathy OA
Cited by 0
Semantic Scholar
Research Topics
Renal Diseases and Glomerulopathies
(37)
Glycosylation and Glycoproteins Research
(11)
Celiac Disease Research and Management
(8)
Chronic Kidney Disease and Diabetes
(7)
Systemic Lupus Erythematosus Research
(6)
Affiliations
University of Alabama
University of Alabama at Birmingham
Middlesex Hospital
Pediatric Nephrology of Alabama