Publication & Citation Trends
Publications
35 total
Linking Amyloid-β and Tau: Amyloid-β Induced Synaptic Dysfunction via Local Wreckage of the Neuronal Cytoskeleton
Cited by 85
OpenAlex
Missorting of Tau in Neurons Causes Degeneration of Synapses That Can Be Rescued by the Kinase MARK2/Par-1 PDF
Cited by 286
OpenAlex
Structural Principles of Tau and the Paired Helical Filaments of Alzheimer’s Disease PDF
Cited by 237
OpenAlex
Swimming against the Tide: Mobility of the Microtubule-Associated Protein Tau in Neurons PDF
Cited by 161
OpenAlex
Protein Kinase MARK/PAR-1 Is Required for Neurite Outgrowth and Establishment of Neuronal Polarity PDF
Cited by 275
OpenAlex
Paullones are potent inhibitors of glycogen synthase kinase‐3β and cyclin‐dependent kinase 5/p25 PDF
Cited by 371
OpenAlex
The Development of Cell Processes Induced by tau Protein Requires Phosphorylation of Serine 262 and 356 in the Repeat Domain and Is Inhibited by Phosphorylation in the Proline-rich Domains PDF
Cited by 152
OpenAlex
Research Topics
Microtubule and mitosis dynamics
(21)
Alzheimer's disease research and treatments
(11)
Neuroscience and Neuropharmacology Research
(3)
Mitochondrial Function and Pathology
(3)
Photosynthetic Processes and Mechanisms
(3)
Affiliations
Max Planck Society
Center of Advanced European Studies and Research
German Center for Neurodegenerative Diseases
Max Planck Institute for Medical Research
Max Planck Institute of Molecular Physiology